Stroke patients are faced with many challenges in recovery. coMra Therapy can be used safely and effectively to aid in neuro-regeneration and restoring lost function.
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COMRA THERAPY FOR STROKES
At a recent conference held in Russia, Dr Anna Klassen (MD) and Dr Arzhan Surazakov (PhD) presented some important information on the treatment of stroke patients, and the significant role that coMra therapy plays.
Firstly – let’s look at what happens in the brain affected by a stroke. A stroke can either be haemorrhagic or ischemic in origin.
A haemorrhagic stroke can be caused by a concussive injury or high blood pressure, or in severe cases, an aneurysm in the brain.
An ischemic stroke occurs when one of the blood vessels becomes narrowed or blocked (thrombus) and the brain does not have enough blood flow (and thus, oxygen), which then causes damage. The effects of the stroke depend on where the damage lies: i.e, in which hemisphere and particular area of the brain. For example, speech can be affected or with more extensive damage, motor function can also be affected. Ischemic strokes tend to occur in older age.
Due to the lack of oxygen being delivered to the parts of the brain affected by the stroke, Adenosine Triphosphate (ATP) molecules cannot be produced. These are the main energy carriers in our body and are vital for overall functioning. It has been noted that there is a 45% drop in ATP levels 3 hours after a stroke occurs. This means that cells are dying.
Ten minutes after low-level laser therapy is applied, the ATP levels in the cells increase by 221%. What this means, is that with the assistance of laser radiation, it is possible to (re)start the cellular process of ATP synthesis in the mitochondria. Or put more simply, cells under critical stress are rescued. Not only does laser radiation raise energy levels, but it also speeds up the entire process, thus enabling new neurons to replace old neurons.
The ideal treatment for stroke patients is a combination of massage and exercise therapy to restore neuron connection, especially for the musculoskeletal system. Added to this come the benefits from coMra laser therapy which aids in restoring neural and cellular regeneration.
coMra therapy combines low-level laser therapy with the healing radiances of colour, magnetism and (in the Delta) ultrasound to treat pain and many other conditions. Read our coMra 101 blog and How it Works: coMra therapy blog for more on how it works.
Research into Low-Level Laser Therapy (LLLT) as an alternative treatment for stroke injury (as well as Traumatic Brain Injury) has been ongoing. In a 2016 study on Transcranial Low-Level Laser (Light) Therapy for Brain Injury, LLLT was shown to “have a neuroprotective effect,1 while regulating several biological processes.2 Light can penetrate several tissues, including both the scalp and skull, and reach into the brain. Several clinical and preclinical studies have shown that this process can lead to an improved recovery from stroke.3
Dr Larry Wallace presented a case study of a 68 year old female patient with early glaucoma, high blood pressure and heart disease, who had suffered a stroke 5 months prior. This had resulted in headaches and loss of visual field. The blind spot was in the left side of her field of vision, corresponding to the area of the stroke in the right side of the brain.
Dr Wallace applied 12 treatments (adapted from Neurology 3 in the User Guide), once per week.
At the final progress check after the 12 treatments, the patient’s visual field was completely restored and her headaches were much less frequent and far less intense. Doctor and patient were both very satisfied with the improvement.
For more details on how to apply the Neurology treatment please refer to the User Guide.
1 Long-term effect of low energy laser irradiation on infarction and reperfusion injury in the rat heart. Yaakobi T, Shoshany Y, Levkovitz S, Rubin O, Ben Haim SA, Oron U. J Appl Physiol (1985). 2001 Jun; 90(6):2411-9.2 The nuts and bolts of low-level laser (light) therapy. Chung H, Dai T, Sharma SK, Huang YY, Carroll JD, Hamblin MR. Ann Biomed Eng. 2012 Feb; 40(2):516-33.3 Treatment of experimentally induced transient cerebral ischemia with low energy laser inhibits nitric oxide synthase activity and up-regulates the expression of transforming growth factor-beta 1. Leung MC, Lo SC, Siu FK, So KF. Lasers Surg Med. 2002; 31(4):283-8.4CoMra Therapy Case Report – Stroke with visual field loss and headaches.